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  Česky / Czech version Anest. neodkl. Péče, 11, 2000, No. 4, p. 180–183
 
Any Notes to the Pathophysiology of Secondary Cerebral Injury 
SKLIENKA P., PETRAŠOVIČOVÁ I., KOLÁŘ L., JAHODA J., HRDLIČKA R., KULA R. 

Anesteziologicko-resuscitačná klinika FNsP Ostrava, prednosta MUDr. R. Kula, CSc.
 


Summary:

       Authors retrospectively analysed morphologic (CT scan) and multimodal monitoring data in a group of 31 patients with cerebral injury. On the initial CT scan cerebral edema was apparent in 79 % cases. In contrast with this observation, in course of 48 hours from injury ICP value over 20 mmHg was found in 5 % and SjO2 value below 60 % was found in 59 % measurements. In SjO2 < 60 % data set significantly lower values of paCO2 and significantly higher occurrence of CPP values below CPPcrit were found comparing to SjO2 > 60 % data set. Analysing causes of CPP fall below CPPcrit 8 % participation of isolated ICP rise over ICPcrit and 67 % participation of MAP fall below MAPcrit were found. On the basis of this analysis authors presume that ICP rise is not a cause but result of secondary cerebral injury.

        Key words: cerebral edema – intracranial pressure
       

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